They investigated whether exercise regulates functional recovery and heat shock protein in drug-induced diabetes. The study was conducted by dividing rats into groups based on level of exercise and diabetic status.
After two weeks, sedentary rats had a sustained hypersensitivity to tactile and heat stimuli while exercising rats had a delayed progress of hypersensitivity. Exercise also suppressed blood glucose levels and body weight loss.
Researchers concluded that progressive exercise training markedly decreases diabetes-associated neuropathic pain.
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